Oncogenic function of p34SEI-1 via NEDD4‑1‑mediated PTEN ubiquitination/degradation and activation of the PI3K/AKT pathway.

نویسندگان

  • Samil Jung
  • Chengping Li
  • Dongjun Jeong
  • Soonduck Lee
  • Jiyeon Ohk
  • Meeyoung Park
  • Songyi Han
  • Jingjing Duan
  • Changjin Kim
  • Young Yang
  • Keun-Il Kim
  • Jong-Seok Lim
  • Young-Sook Kang
  • Myeong-Sok Lee
چکیده

A 34-KD protein encoded by the SEI-1 gene (p34(SEI‑1)), is a relatively recently discovered oncoprotein that has multiple important biological functions. Our data show that p34(SEI-1) enhances cancer cell survival and promotes tumorigenesis by downregulating the tumor suppressor PTEN, a negative regulator of the PI3K/AKT signaling pathway, and therefore activating the PI3K/AKT signaling pathway. In this process, p34(SEI-1) positively affects NEDD4-1 gene expression both at the transcriptional and protein levels. Furthermore, the expression levels of p34(SEI-1) and NEDD4-1 were found to be coordinated in tumor tissues obtained from patients with breast cancer. We also show that p34(SEI-1) affects the subcellular localization of PTEN.

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عنوان ژورنال:
  • International journal of oncology

دوره 43 5  شماره 

صفحات  -

تاریخ انتشار 2013